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Andrew S. Resnick, M.D., M.B.A.
Gastrointestinal Surgery
 
Research
Hospital of the University of Pennsylvania, Department of Surgery, Division of Surgery Education 2003-present
  • Ongoing research to examine interaction of education, efficiency, and patient safety. In separate projects, studied operating room data, faculty teaching scores, hospital finance data, patient satisfaction data, resident education and incident report data. Determined relationships between teaching scores and Relative Value Units (RVUs), department and hospital margin for different specialties, patient satisfaction and surgical house staff, and looked at patterns associated with resident incident reports.
 
Howard Hughes Medical Insitute-National Institutes of Health Research Scholars Program - 1 year fellowship in Bethesda, MD from 8/97-6/98.
  • Worked in Dr. Warren Strober's laboratory in the Mucosal Immunity Section, National Institute of Allergy and Infectious Disease. Studied Th1 cell development and the effects of TGF- on naive CD4+ T cells and differentiated CD4+ Th1 cells and Th2 cells. Demonstrated that TGF- acts to suppress Th1 cell proliferation and IFN-gamma production by inhibiting the phosphorylation of an intracellular signal transduction protein, STAT4.
 
Yale University, Department of Cellular and Molecular Physiology - Summer, 1996.
  • Analyzed blood samples from patients with non-Zollinger Ellison Syndrome hyper gastric acid secretion. Extracted DNA and used PCR to look for mutations in the endocytosis signalling region of the subunit of gastric H,K-ATPase.
 
Beth Israel Hospital, Department of Surgery - Summers, 1994, 1995.
  • Conducted research on intestinal epithelial cell ion transport in the laboratory of Jeffrey Matthews, M.D. at the Beth Israel Hospital in Boston. Techniques included electrophysiological and fluoroscopic analyses.
 
Presidential Scholar and Honors Thesis in laboratory of C.R. McClung, Ph.D. - 1992-1995.
  • Research in molecular genetics to quantify the photic and non-photic gene regulatory elements in the plant Arabidopsis. Ran genetic tests to determine the circadian expression of different kinds of mRNA. Mutated plants through the addition of noxious gases to isolate mutants in circadian regulation.
 


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